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Volume 5, Issue 5, pp. 550-563
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History
- Received December 30, 2013
- Revision received January 27, 2015
- Accepted January 27, 2015
- Published first February 4, 2015.
Article Versions
- Previous version (February 4, 2015 - 07:06).
- Previous version (April 20, 2015 - 07:13).
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©2015 American Association for Cancer Research.
Author Information
- Andrea Lunardi1,
- Shohreh Varmeh1,
- Ming Chen1,
- Riccardo Taulli1,
- Jlenia Guarnerio1,
- Ugo Ala1,
- Nina Seitzer1,
- Tomoki Ishikawa1,
- Brett S. Carver2,
- Robin M. Hobbs1,
- Valentina Quarantotti1,
- Christopher Ng1,
- Alice H. Berger1,
- Caterina Nardella1,
- Laura Poliseno1,
- Rodolfo Montironi3,
- Mireia Castillo-Martin4,
- Carlos Cordon-Cardo4,
- Sabina Signoretti5,6 and
- Pier Paolo Pandolfi1,*
- 1Cancer Research Institute, Beth Israel Deaconess Cancer Center, Department of Medicine and Pathology, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, Massachusetts.
- 2Cancer Biology and Genetics Program, Sloan Kettering Institute, Division of Urology, Department of Surgery, Memorial Sloan Kettering Cancer Center, New York, New York.
- 3Institute of Pathological Anatomy and Histopathology, Polytechnic University of the Marche Region (Ancona), United Hospitals, Ancona, Italy.
- 4Department of Pathology, Mount Sinai School of Medicine, New York, New York.
- 5Department of Pathology, Brigham and Women's Hospital, Harvard Medical School, Boston, Massachusetts.
- 6Department of Medical Oncology, Dana-Farber Cancer Institute, Harvard Medical School, Boston, Massachusetts.
- ↵*Corresponding Author:
Pier Paolo Pandolfi, Cancer Research Institute, Beth Israel Deaconess Cancer Center, Departments of Medicine and Pathology, Beth Israel Deaconess Medical Center, Harvard Medical School, 330 Brookline Avenue, Boston, MA 02215. Phone: 617-735-2121; Fax: 617-735-2120; E-mail: ppandolf{at}bidmc.harvard.edu
A. Lunardi and S. Varmeh contributed equally to this article.
May 2015
Volume 5, Issue 5
Volume 5, Issue 5
Suppression of CHK1 by ETS Family Members Promotes DNA Damage Response Bypass and Tumorigenesis
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