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Volume 6, Issue 7, pp. 754-769
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History
- Received November 19, 2015
- Revision received May 4, 2016
- Accepted May 4, 2016
- Published first May 6, 2016.
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- Previous version (May 6, 2016 - 06:22).
- Previous version (June 20, 2016 - 07:41).
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©2016 American Association for Cancer Research.
Author Information
- Hidenori Kitai1,2,
- Hiromichi Ebi1,3,*,
- Shuta Tomida4,
- Konstantinos V. Floros5,
- Hiroshi Kotani1,
- Yuta Adachi1,
- Satoshi Oizumi2,
- Masaharu Nishimura2,
- Anthony C. Faber5 and
- Seiji Yano1,*
- 1Division of Medical Oncology, Cancer Research Institute, Kanazawa University, Ishikawa, Japan.
- 2First Department of Medicine, Hokkaido University School of Medicine, Hokkaido, Japan.
- 3Institute for Frontier Science Initiative, Kanazawa University, Ishikawa, Japan.
- 4Department of Biobank, Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University, Okayama, Japan.
- 5VCU Philips Institute for Oral Health Research, School of Dentistry and Massey Cancer Center, Virginia Commonwealth University, Richmond, Virginia.
- ↵*Corresponding Authors:
Hiromichi Ebi, Institute for Frontier Science Initiative, and Division of Medical Oncology, Cancer Research Institute, Kanazawa University, 13-1, Takaramachi, Kanazawa, Ishikawa 920-0934, Japan. Phone: 81-76-265-2794; Fax: 81-76-234-4524; E-mail: hebi{at}staff.kanazawa-u.ac.jp; and Seiji Yano, E-mail: syano{at}staff.kanazawa-u.ac.jp
H. Kitai and H. Ebi contributed equally to this article.
July 2016
Volume 6, Issue 7
Volume 6, Issue 7
Epithelial-to-Mesenchymal Transition Defines Feedback Activation of Receptor Tyrosine Kinase Signaling Induced by MEK Inhibition in KRAS-Mutant Lung Cancer
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